Advances in Laboratory Diagnosis and Clinical Management ofGilbert Disease :A Comprehensive Review

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dc.contributor.authorNkiruka R Ukibe
dc.contributor.authorChioma Theresa Onwe
dc.contributor.authorC.E. Onah Ezinne G Ukibe
dc.contributor.authorBlessing C.Ukibe
dc.contributor.authorVictory Ezennia Ukibe
dc.contributor.authorEmmanuel Ifeanyi Obeagu
dc.date.accessioned2024-06-21T11:45:11Z
dc.date.available2024-06-21T11:45:11Z
dc.date.issued2024-06-01
dc.description.abstractGilbert’s syndrome (GS) is an extensively mild condition characterized by periods of elevated levels of bilirubin in the blood. The bilirubin is an orange yellowish tinted molecule which is produced when red blood cells are broken down and this substance is eliminated from the body only after it undergoes bio-transformation in the liver which converts unconjugated bilirubin to conjugated bilirubin. When the level of unconjugated bilirubin increases beyond a determined point, the bilirubin pigment starts to discolour the cornea of the eyes (making them to appear light yellow) and with higher levels the skin may also turn to yellow (jaundice). Gilbert’s syndrome also known as constitutional hepatic dysfunction and familial non haemolytic jaundice. People with Gilbert’s syndrome have an inherited abnormality that causes reduced production of an enzyme involved in processing of bilirubin. A
dc.description.sponsorshipKampala International University, Uganda
dc.identifier.issn2736-7319
dc.identifier.urihttp://hdl.handle.net/20.500.12493/14484
dc.language.isoen
dc.publisherIAA Journal of Scientific Research
dc.titleAdvances in Laboratory Diagnosis and Clinical Management ofGilbert Disease :A Comprehensive Review
dc.title.alternative
dc.typeArticle
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